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The first study, performed by allen et al We aimed to evaluate the effect of repetitive endovascular intraarterial (ia) combined milrinone and nimodipine administration as rescue therapy for severe refractory cerebral vsp. [8] in 1983, suggested that nimodipine could affect outcome, but the trial was small and largely a selection of patients with cerebral ischemia from cerebral vasospasm.
Deuterated and thus structurally different nimodipine was added to all samples before analysis for internal standardization and optimization of validity Intraarterial (ia) milrinone and nimodipine were suggested as safe treatment options Nimodipine in serum samples was evaluated based on protein precipitation
Csf and microdialysates were assayed directly.
Our results show that nimodipine reduces the formation of microvasospasms, thus, shedding new light on the mode of action of a drug routinely used for the treatment of sah for >3 decades In clinical practice, nimodipine is used to control cerebral vasospasm (cvs), which is one of the major causes of severe disability and mortality in patients with aneurysmal subarachnoid hemorrhage (asah). Purpose delayed cerebral ischaemia (dci) and cerebral vasospasm (cvs) remain major causes of poor outcome in survivors of aneurysmal subarachnoid haemorrhage (asah) We aimed to investigate the safety and efficacy of intracisternal administration of nimodipine in patients suffering from symptomatic cvs refractory to treatment with induced hypertension and endovascular vasodilator therapy.
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